How do we see colours?
Our ability
to perceive colours is one of the things that makes the world beautiful. Colours
are not only beautiful they are also exceptionally useful. The colour of an
apple lets you know whether it is ripe or not. Colourful displays, book and
magazine covers attract your attenton. Colours are also essential to fashion.
Each season has a new set of colours to wear. Colour
perception deficiency affect almost 8% of the male population. It is believed
to be inherited usually for the maternal grandfather. The typical red-greed
colour perception deficiency can be greatly improved by Vision Training. Read
on a learn more about colour perception and how it can be improved. The scientific
study of colour starts with Newton's great work, the Optick (1704).
This is an extraordinary work detailing Newton's experiements in Trinity College,
Cambridge. The Optick contains Newton's most speculative thoughts
on physics and man's relation through perception of the Universe. Newton showed
that white light is made up of all the spectral colours. The later wave theory
made it clar that each colour corresponds to a specific frequency. Newston's
original sketch of an experiment with colour. He split sunlight into a spectrum
through a large prism. The spectrun is again recombined into white light
through a second prism. Colour
perception is our ability to see a range of spectral wavelengths from 380
nm Violet to 780 nm Red. As the wavelength change the colour perception
changes. Red and Blue light combine into Violet. Red and Green light combine
into Yellow and Blue and Green light become Turquise. Thomas
Young (1773 - 1829) proposed the trichomatic theory (1802) which suggest that
there are three primary colours only. German scientist Herman von Helmholtz
developed Young's ideas further and it became the Young-Helmholtz theory.
There are three colour sensitive receptors (cone cells) which respectively
responds to red, greed and blue. All colours are seen by a mixture of signals
from the three colour receptors. Incidentally, this is the same principle
behing your computer screen. In computer parlance it is known as the RBG colour
system. Hue
discrimination The hue
defines the smallest wavelength of light with the smallest diffrence that
is possible to diffrentiate by the human eye. We notice that there is a dramatic
increase in the red-green sensitivity producing exceptional hue discrimination
ability in the gree - yelloe - orange - red region. This is a very useful
feature because various foods and fruits display their readiness to be eaten
by their colour change. You can instantly see whether an apple is ready to
eat or not by its colour. Colour
film is essentially a complex arrangement of the three primary colour filters.
Kodachrome slides are able to reproduce all the complexity of colour seen
in nature. Colour
vision is very complex and cannot be reduced to simple theories. Colour depends
not only on the wavelengths and intensities, but also on differneces on intensities
between regions, and whether the patterns are accepted as representing known
objects. This involves high-level processing in the brian, which is extremely
difficult to investigate. The eye
tends to accept as white not a particular mixture of colours, but rather the
genreral illumination. Thus you see a car's headlights as white while on a
country drive, but in town where there are bright white lights for comparison,
they look quite yellow, and the same is true of candle light. This means that
what is takes as reference for white can shift. Expectation and knowledge
of the normal colour is an impportant factor in colour perception. Colour
perception deficiency Surprisingly,
the common confusion where red is confused with green, was not discovered
before late eighteenth century when chemist John Dalton found that he could
not distinguish certain substances by their colour although others could do
so easily. Colour
vision tests depend on isolating colour as the one identifying characteristic,
and then it is easy to show whether a person has normal ability to distinguish
between colous, or whether he or she sees a single colour what to others appear
different colours. It is most common to find a reduced sensitivity to some
colours rather than a complete absense of colour. The properties
of red and green light required to match a monochromatic yellow is the most
important measure of colour deficiency. Lord Rayleigh discovered in 1881 that
people who confuse red with green, require a greater intensity of either red
or green to match yellow. A special instrument has been developed for testing
this colour deficiency, it is called an anomaloscope. This insturiment
takes advantage of the fact that yellow is always seen as a mixture of red
and green. The reason
for red-green colour perception deficiencies is not clear. However, experiements
with the anomaloscope shows that colour anomaly cannot be due to colour adaptation.
The general belief is that red-green colour perception deficiency is supposed
to be a reduction in the sensitivity of one or more colour receptors of the
retina, through partial loss of photo-pigment. There may be many causes, but
it is certainly not due to shortage of photo-pigment or the anomaloscope would
not work. The common
red-green colour perception deficiency is more likely to be an interpretation
of sensory data provided to the visyal cortex processing colour vision. German
physiologist Edward Hering noted that red and green are never seen simultaneously
also the colour red/green is never used. Colours are either red or green,
but not both. The same is true for blue and yellow. This observation lead
Hering to propose the opponent colour vision theory (1920 / 1964). In the
late 1950’s Leo Hurvich and Dorothes Jameson provided quantitative
date in support of the notion that colour opponency plays an important role
in processing colour information. They used a hue cancellation procedure (hue
is added to the stimulus until it turns white) to determine spectral sensitivities
of the red-green and blue-yellow opponent channels. The discovery
of colour opponent neurons in the visual system tells us that the recetoral
information (trichomacy)) is coded in an opponent fashion at post-receptoral
levels. In other words the three classes of colour sensitive cone cells are
“wired” together in such as way that they are spectrally antagonistic.
The process occurs in the retina and the level of horizontal cells. In summary,
primary colour vision is thrichromatic, with this trichormatic signal encoded
in an opponent fashion. This opponent processing occurs very early in the
visual system, at the level of horizontal cells. It is scientifically established
that hue information is encoded by red-green and blue-yellow neurons. It is
not clear whether brightness information is encoded by these neurons or by
a separate class of non-colour opponent cells

It is believed that the red-green and blue-yellow channels only code hue information.
The brightness is presumably coded by an independent brightness channel. If
the frequency of action potential is plotted as a function of wavelength.
We see that low wavelength stimuli (below 550 nm) cause inhibition, or a decreased
firing rate from this cell.
In contrast, long-wavelength stimuli (longer than 550 nm) produce excitation,
or an increased rate of neural firing. When a photosensitive cell responds
to one portion of the spectrum with excitation and another with portion with
inhibition, it is referred to as colour opponent neuron.
